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showed that Se boosted GPX1 expression by inhibiting DNMT2-mediated DNA methylation of the GPX1 promoter from lessening ROS production in advanced glycation end-product (AGE)-induced heart failure [114]

Despite these concerns, the overall risk of bias was low for the majority of the included RCTs, suggesting that the findings of these studies provide robust evidence for the efficacy and feasibility of VR-based interventions in individuals with SCD, or MCI

Beyond redox regulation, GSH is involved in several essential cellular processes, including the synthesis of DNA and proteins, modulation of enzyme function, gene expression regulation, signal transduction, and both intracellular and transmembrane transport [13]